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Biologic
RCSB PDB 5WUX

Certolizumab pegol

Cimzia
Mechanism of action
TNF-alpha (PEGylated )
In plain language
A lab-made fragment, chemically stabilized with a polymer coating, that grabs onto TNF-alpha — a protein that fuels inflammation — and stops it from triggering the immune system.
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How it works
Certolizumab pegol is a recombinant, humanized Fab' fragment directed against human TNF-alpha, conjugated to a polyethylene glycol (PEG) moiety. It binds and neutralizes both membrane-associated and soluble TNF-alpha, blocking downstream pro-inflammatory signaling. Because it consists only of the Fab' -binding fragment and lacks an Fc region, it does not fix complement or mediate antibody-dependent cell-mediated cytotoxicity, distinguishing it mechanistically from full-length IgG anti-TNF antibodies such as adalimumab. PEGylation increases the molecule's serum half-life, allowing less frequent dosing.
Therapeutic applications
FDA-approved for moderately to severely active rheumatoid arthritis, active polyarticular juvenile idiopathic arthritis, active psoriatic arthritis, active ankylosing spondylitis and non-radiographic axial spondyloarthritis, moderate-to-severe plaque psoriasis, and Crohn's disease with moderately to severely active disease.
Class
Fab fragment
Therapeutic area
Immunology
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